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GREEN TEA,
FLUORIDE AND THE THYROID
OPEN LETTER TO: Susan Cameron-Block
Host - Current Health Issues
August 24, 1999
Dear Susan,
I am writing this letter with the intent to inform on
various issues associated with the use of fluorides, especially as
it relates to green and black teas, and to voice our concern about
the continued promotion of green tea as a drink "beneficial to
one's health" on your radio show "Current Health
Issues".
Tea is very high in fluoride content. Fluoride in tea is much
higher than the Maximium Contaminant Level (MCL) set for fluoride in
drinking water.
Tea leaves accumulate more fluoride (from pollution of soil
and air) than any other edible plant (1,2,3). Fluoride content in
tea has risen dramatically over the last 20 years, as has tea
consumption (4).
While in 1976 a Belgian analysis showed content of between 50
and 125 ppm fluoride in 15 varieties of tea (3), a Polish study in
1995 found fluoride content of up to 340 ppm in 16 varieties of
black tea (5). A major Canadian study published in 1995 reports
average fluoride content in tea to be 4.57 mg/l in the 1980's.(6)
A website by a pro-fluoridation infant medical group lists a
cup of black tea to contain 7.8 mgs of fluoride (7), which is
roughly the same amount as if one were to drink 7.8 litres of water
in an area fluoridated at 1ppm. It is well known that fluoride
in tea gets absorbed by the body similarly as the fluoride in
drinking water (1,8).
Some British and African studies from the 1990's showed a
daily fluoride intake of between 5.8 mgs and 9 mgs a day from tea
alone.(9,10,11)
In order to understand a dose/concentration relationship
properly, one needs to realize that the level of fluoride at 1
part-per-million (ppm) = 1 mg/l was set in the 40's when TOTAL
intake was considered to be only about 1 mg/day in areas with
fluoridated water. It was thought that the fluoridation of water
supplies at 1 ppm (1 mg/l) would duplicate this intake, assuming
that people would drink 4 glasses of water a day. However, average
current total intake of fluorides is approaching the 8mg/day range,
according to the last official data available from the US PHS (1991)
and other publictions (12).
TOTAL intake from ALL sources is the amount to be considered for any
adverse health effect evaluation. (13,14,15)
The fact that fluorides accumulate in the body is the reason
why a MCL for fluoride content in water needs to be set by the US
Environment Protection Agency (EPA) - by law under the US Surgeon
General. This is to be done specifically to avoid a condition known
as Crippling Skeletal Fluorosis (CSF). The MCL is set so as to only
avoid the third and crippling stage of this disease. It is set at
4ppm => 4mg/liter, assuming that people will retain half of this
amount (2mg), and therefore be at a "safe" level. The EPA
scientists, whose job and legal duty it is to set the MCL, declared
that this level was set fraudulently by outside forces, and that 90%
of the data showing the mutagenic properties of fluoride were
omitted. (16)
Virtually every company selling
green tea advertises it's high fluoride content as
"beneficial" in preventing cavities, promulgating the
misleading and false data supplied for the last 50 years by the ADA/CDA
and other dental health trade organizations, as well as various
public health agencies. There are NO double-blind studies anywhere
proving the efficacy of fluoride as a caries preventative (17).
There ARE double-blind studies proving adverse health effects, at
the level of 1ppm (1mg/l) in water.(18) There are no studies
documenting safety at any intake level..
THYROID MEDICATION:
Drinking a cup of tea with fluoride content as
mentioned above (7.8mg) would mean a fluoride intake much higher(!)
than amounts which were actually given as medication to treat
hyperthyroidism (-> over-functioning thyroid) for numerous
decades - in several countries - specifically to reduce thyroid
activity! [(2 -10 mg NaF/day => 0.9mg - 4.5mg F-)] (19,20,21,22)
In the 1930's May reported having _successfully_ treated
1,158 hyperthyroid patients within 6 years with either sodium
fluoride or fluorothyrosine, given per mouth. Among products later
released on the market were Pardinon and Tyrosin (23, 24).
Checking an older Merck Index
will verify this information.(25) Gorlizer von Mundy treated
patients for more than 30 years in baths containing HF (30ccHF in
200 l water). Later fluorides were deemed not "reliable
enough" to be recommended as an antithyroid (26).
RE: CANCER AND GREEN TEA
While there can be no doubt as to the beneficial
effects of individual anti-oxidants found in green tea, the same
cannot be said about green tea as a beverage. Existing studies tend
to concentrate on active ingedients of green tea, such as
epigallocatechin gallate (EGCG), a compound that belongs to a family
of anti-oxidants known as polyphenols. EGCG and other polyphenols
are constituents of tea - especially of green tea. However, no
studies exist investigating the effects of fluorides on these
anti-oxidants. Existing studies involving other anti-oxidants and
fluoride compounds give evidence that fluorides can adversely affect
the action of anti-oxidants(27). Thus, while isolated anti-oxidants
may slow down the development of some forms of cancer in
experimental studies, their effect may be annihilated in their
complex natural environment (as a sum of the action of all the
substances present).
Several reviews of available data seem undecided in their
conclusions as to the inhibition of carcinogenesis in experimental
animals by tea or tea compounds. Data reviewed by Blot et al. (28)
suggest "at most a modest benefit, since there is considerable
international variation in tea consumption but generally small
differences in cancer rates...More relevant case-control and cohort
studies show mixed results."
Other epidemiological and human studies have also shown
varying results. In a review by Bushman (29) thirty-one human
studies and four reviews were examined. Among five studies reporting
on colon cancer, three found an inverse association and one reported
a positive association. For rectal cancer, only one of four studies
reported an inverse association; increased risks were seen in two of
the studies. An inverse association was suggested for urinary
bladder cancer in two of two studies. While lung cancer studies have
shown an inverse effect with Okinawan tea, a tentatively increased
risk was shown in another study, clearly indicating that more
research into this matter is needed. In a recent study on Finnish
men, published in 1998 by Terryl Hartman and others, again a
positive correlation between colon cancer and tea intake was found.
Colon cancer occurrence increased with higher intake (30).
Many available green tea/cancer studies last only a few
months, and do not take into account the cumulative effects of
fluoride, which is a known cancer promoter, and has the ability to
transform healthy cells into cancerous ones. (1,17,35,36) For any
conclusive evidence to be obtained this must be considered, for long
time fluoride ingestion has been shown to _cause_ cancer, especially
osteosarcomas and uterine cancer. (31,32)
Dean Burk, for many decades Chief
Chemist at the National Cancer Institute, testified at congressional
hearings in 1981 stating that over 40,000 cancer deaths in that year
were attributable to fluoridation (33). He has said that no chemical
causes as much cancer, and faster, than fluorides (34). Public
health officials are quick to say that this data is not verified,
which is entirely untrue, for international research as well as
congressional hearings and court proceedings HAVE verified this
information. (1,2,16,17,31,32,33,34,35,36,37,38)
TEA AS A SOURCE OF FLUOROSIS
Dental fluorosis (mottled teeth) is the first _visible_ sign
of fluoride poisoning.
Studies conducted on tea consumption in Tibetan children by
Cao et al. found both dental (51.2%) and skeletal (32.83%) fluorosis,
mainly as a result from drinking brick tea, also known as milk tea
(39). More studies by Cao and others reported similar results
(40,41) as did a study from Chile showing dental fluorosis risks in
22.1% of the children consuming tea as a main beverage (42).
Many similar studies on tea as well as other beverages have been
published in the journals of the American Dental Association (ADA)
or American Medical Association (AMA) themselves.
Studies on hydrofluoric-acid workers
from an electronics company documented that, among the influences of
fluorine-containing foodstuff on fluoride content in the biological
fluids, the effect of black tea and/or green tea intake was
"particularly remarkable". Measuring the urine and serum
levels of fluorine ion, in the case of the
non-hydrofluoric-acid workers, the concentration increased to about
double of the control value. Similarly in a diet test on volunteers,
the concentration increased about six times. (43)
There are several other factors to
consider regarding fluoride content in tea. One is the amount of
fluoride leeching over time. Chinese teas continue to release F-
throughout the first hour of infusion, whereas release of F- from
Ceylon/Indian teas is essentially completed after 5 minutes.(44)
The first study to investigate
fluoride content in de-caffeinated teas found an even higher
fluoride content in those teas as compared to their caffeinated
counterparts. (45) It is thought that this is due to the high
fluoride content in the water involved in the de-caffeination
process, which then would also make coffee similarly de-caffeinated
high in fluoride content.
In addition, the caffeine in
tea has a great augmentative effect on the bio-availability of
fluoride. In 1990 researchers at the University of Texas even
theorized that "the rise in incidence of dental fluorosis in
North America is mainly due to the replacement of water intake by
caffeine-containing beverages among the young population".(46)
In 1990 German researchers wrote that "continuous intake of
black tea rich in fluorides leads to distinct increase of fluoride
content of temporary teeth. This is to consider analogous a caries
prophylaxis."(47)
Considering this, and tea market
satistics which report that, "on any given day, nearly 127
million people -- half of all Americans -- are drinking tea",
and that tea is available in 80% of US households (4), one must
seriously ask why anyone in their right mind would want to add to
the already existing load by adding fluorides to the public water
supply.
FLUORIDE AND ALUMINUM IN TEA
To make matters much worse for human health, fluorides
in teas are found together with aluminum. The combination of
aluminum and fluorides in tea is of urgent concern, due to the
increased damage done by fluorides when in the presence of aluminum,
especially neurological and renal damage)(17).
A study by Wei and others
reported a high correlation (r = 0.81) found between the released F
and Al in all tested Chinese, Indian and herbal teas.(48)
Nabrzyski and Gajewska (49)
report: "..In the 16 samples of commerically
available brands of black teas, the levels of aluminium and fluoride
ranged from 445 to 1552 ppm (mean = 897 +/- 264 ppm) and from 30 to
340 ppm (mean 141 +/- 85 ppm), respectively. In six herbal teas, the
mean levels of aluminium and fluoride were lower, and amounted to
218.9 +/- 150.7 ppm and 6.0 +/- 6.9 ppm, respectively..."
That the aluminum present is
indeed resorbed in the simultaneous presence of fluoride is shown in
a study by Drs. Klaus R. Koch and Colleagues at the University of
Cape Town. They examined the urinary excretion of aluminum (which is
an indicator of its resorption) in healthy male volunteers after
drinking equal volumes (1.2 litres) of tea, coffee or tap water on
separate days. In _every_ case the amount of aluminum excreted over
the 12-hour period increased on the day when tea was taken. Their
results indicate that tea consumption must be considered in any
assessment of the total dietary intake of aluminium in human
beings.(50)
A most important study from
1998 conducted at the Nanchang University in China showed that in
older rats fed green tea water extract or green tea leaves, the
cerebrum calcium contents were significantly decreased and aluminum
contents increased. Zinc contents in the cerebrum were also
gradually decreased with the increase of tea leaves dose and tea
concentration(51). The cerebrum is the portion of the brain (frontal
lobes) where thought and higher function reside.(52)
The fluoride/aluminum association is
of particular importance as it relates to Alzheimer's Disease.
Aluminum by itself is not readily absorbed by the body. However, in
the presence of fluoride ions, the fluoride ions combine with the
aluminum to form aluminum fluoride, which is absorbed by the
body. In the body, the aluminum eventually combines with oxygen to
form aluminum oxide or alumina (53). Alumina is the compund of
aluminum that is found in the brains of Alzheimer's disease. In the
brain, protein binds to the alumina, and "that is the key to
the plaques and tangles which are the hallmarks of this terrible
disease" (54). In a study by Dr. Robert Isaacson at the
State University of New York, aluminum fluoride was added to the
rats diet. This, contrary to normal expectations, passed through the
brain barrier and gave the rats short term memory, smell sensory
loss, unsteady gait, and loss of structures of the neo-cortex and
hippocampus, all symptoms of Alzheimer's.(53,54,55,56) A Varner
and Jensen study conducted with Isaacson confirmed this in 1998.(57)
Free fluorine ions and
traces of aluminum form a complex, fluoroaluminate, which stimulates
cellular G proteins. Such a complex can form in food, drinking
water, in the organism after fluoride ingestion or absorption, or
after adminstration of a vaccine. Susa (58,59) reports that "fluoroaluminate
crosses the cell membrane and directly binds to the
membrane-associated inactive Ga protein subunits. Within the Ga
subunit, fluoroaluminate occupies the position next to GDP. The
resulting Ga-GDP-AlF4- complex assumes an active state conformation,
which resembles that of Ga-GTP complex. Under physiological
conditions, Ga-GTP complex is formed upon activation of seven
transmembrane receptors that couple to heterotrimeric G
proteins...Both fluoroaluminate-activated and receptor-activated Ga
subunits are capable of transmitting intracellular signals that lead
to cellular responses."
There are hundreds of G
protein-coupled receptors. (60) The thyroid stimulating hormone (TSH)
receptor is also coupled to the G protein. The TSH receptor is
densely expressed in the thyroid gland and mediates the production
and secretion of thyroid hormones. (61) To presume that the
fluoroaluminate will not interfere here is simply naive.
There have been hundreds of
scientific studies using aluminum/fluoride complexes in the last ten
years. A review of the literature by Strunecká and Patocka reveals
that aluminofluoride complexes influence all cells and tissues of
the human body with "powerful pharmacological
efficacy."(62,63)
[This MEDLINE search will return approx.
100 fluoroaluminate-related items:] http://www.ncbi.nlm.nih.gov/htbin-post/Entrez/query?db=m&form=6&uid=99
17518&dopt=m&dispmax=20
NEUROLOGICAL EFFECTS OF FLUORIDE
Other numerous studies in the late 1990's have been
published documenting the effects of fluoride on the neurological
system.(65,66,67,68,69)
They are briefly
addressed here in an excerpt from a paper published by the National
Treasury Employees Union (NTEU) Local 280, formerly National
Federation of Federal Employees Local 2050, representing the
approximately 1500 scientists, lawyers, engineers and other
professional employees at EPA Headquarters in Washington, D.C.:
"Why EPA'S Headquarters Union
of Scientists Opposes Fluoridation"
Issued May 1, 1999 (17):
"In 1995, Mullenix and
co-workers showed that rats given fluoride in
drinking water at levels that give rise to plasma fluoride
concentrations in the range seen in humans suffer
neurotoxic effects that vary according to when the rats were given
the fluoride - as adult animals, as young animals, or through the
placenta before birth. Those exposed before birth were born
hyperactive and remained so throughout their lives. Those exposed as
young or adult animals displayed depressed activity. Then in 1998,
Guan and co-workers gave doses similar to those used by the Mullenix
research group to try to understand the mechanism(s) underlying the
effects seen by the Mullenix group. Guan's group found that several
key chemicals in the brain - those that form the membrane of brain
cells - were substantially depleted in rats given fluoride, as
compared to those who did not get fluoride.
"Another 1998 publication by Varner, Jensen and others reported
on the brain- and kidney damaging effects in rats that were given
fluoride in drinking water at the same level deemed
"optimal" by pro-fluoridation groups, namely 1 part per
million (1ppm). Even more pronounced damage was seen in animals that
got the fluoride in conjunction with aluminum. These results are
especially disturbing because of the low dose level of fluoride that
shows the toxic effect in rats - rats are more resistant to fluoride
than humans. This latter statement is based on Mullenix's finding
that it takes substantially more fluoride in the drinking water of
rats than of humans to reach the same fluoride level in plasma. It
is the level in plasma that determines how much fluoride is 'seen'
by particular tissues in the body. So when rats get 1 ppm in
drinking water, their brains and kidneys are exposed to much less
fluoride than humans getting 1 ppm, yet they are experiencing toxic
effects. Thus we are compelled to consider the likelihood that
humans are experiencing damage to their brains and kidneys at the
'optimal' level of 1 ppm."
("Optimum intake" = 1mg/day)
Toothpaste also contains a significant quantity of Al,
more so, when packed in Al tubes. (70) That children frequently
ingest too much toothpaste is well established and the reason why
since April 1997 a poison warning is to be placed on all
fluoride-containing toothpastes in the US. It is an absolute
disgrace that this is not the same in Canada, especially when the US
FDA has issued several Import Alerts and customs detention orders,
documenting fluoride amounts double that of permissable content
originating in Canada! (71)
THYROID HORMONES
Thyroid hormones are extremely
important in the regulation of metabolic processes and brain
development. Every cell in the body depends upon thyroid hormones
for regulation of their metabolism.
Many of the symptoms
documented in the vast literature on the subject of chronic or
low-grade fluoride poisoning can be directly related to thyroid
functions and disorders. One of the most prominent features of
preskeletal fluorosis is the extraordinary general fatigue
experienced by most sufferers, a marked weakness usually linked to
low activity of the thyroid gland. (2) This has been reported since
the classic 1930's Roholm study on cryolite workers exposed to
fluorides, a study which still serves as the basis for occupational
fluoride exposure regulations. (73) At the time of Roholm's work the
specialized field of "endocrinology" was yet to be
recognized as a reputable discipline. Thyroid diseases were poorly
understood. From 1940 to 1970, the application of radioiodine
improved this understanding immeasurably. Fragu (74)
writes:"The main transformations brought about by this tool
were the knowledge of radioiodine uptake mechanisms, basis of its
therapeutic effect, complete identification of thyroid
hormonosynthesis, serum transport of thyroid hormones and thyroid
imaging. More recently immunological and molecular paradigms changed
the understanding of thyroid diseases."
It is only in the last two decades
during which endocrinology has progressed so rapidly, that now over
150 symptoms and associations can be identified in hypothyroidism.
Almost all(!) correlate with known symptoms of fluoride
poisoning.(74) Most of the double-blind test results of fluoride
poisoning found in Moolenburgh's study on water containing 1ppm of
fluoride - which led to the ban of fluoridation in Holland - are now
recognized symptoms of hypothyroidism. (75) The effects of fluoride
on the thyroid gland have been studied so extensively, that it
baffles the mind how experts on thyroid disease from Harvard or the
University of Toronto can claim that fluorides do not affect thyroid
gland function, especially when it has been used as medication to do
just that! (76) This stance just defies all knowledge properly
gained in the last 70 years of related research. One cannot
find any mention of fluorides in ANY current "official"
thyroid disease related literature. And this at fluoride intake
levels and at dental fluorosis rates as high as they are!
Already in 1940 authors Robert H.
Wilson and Floyd DeEds from the United States Department of
Agriculture (discussing the role of fluorine in
pesticide sprays), wrote:
"Should a spray residue
tolerance limit for fluorine be set to protect the normal, the
hyperthyroid, or the hypothyroid individual? ... should the
tolerance limit take into consideration that in certain areas the
public is already exposed to a fluorine intake in the drinking
water?"(77)
We have posted over 100 studies
documenting the adverse effects of fluoride on the thyroid gland
from the last 70 years or so in the Virtual Library on Fluoride
Research (78) at:
http://www.bruha.com/fluoride/html/thyroid_studies.htm
THYROID & SIDS/DOWN SYNDROME
A toxicologist in the United
Kingdom recently found that perinatal deaths in a fluoridated area
was 15% higher than in neighboring non-fluoridated areas. The
fluoridated area had a higher socio-economic status and would have
been
expected to have less perinatal deaths. The fluoridated area also
had a 30% higher rate of Down's Syndrome. (79a) Down's Syndrome is a
disease associated with thyroid pathology.(79b) Chile banned
fluoridation because of research by the world-reknowned researcher
and Nobel price winner, Dr Albert Schatz, which showed a link to
infant deaths due to fluoridation.(80) Already in the 1950s, Ionel
Rapaport published studies showing links between Down's Syndrome and
natural fluoridation.(81)
[In this context an article should
be noted which appeared in the October1995 issue of the
"Monitor", a publication by the American Psychological
Association, which reported of the similarity in neurological signs
in Down's Syndrome and Alzheimer's disease. The link between the two
dates back to the 1940s when George Jervis, who later became the
first director of New York State Institute for Basic Research in
Developmental Disabilities, conducted autopsies on people with
Down's syndrome and found they had the same neuropathology as people
with Alzheimer's disease. People with Down's syndrome tend to age
faster than the general population and suffer a wide range of
accompanying health problems--many of which mimic or mask the
presence of Alzheimer's disease.(82)]
THYROID AND LEARNING DISORDERS
Learning disorders such as
Attention Deficit Hyperactivity Disorder (ADHD) did not knowingly
exist before the fluoridation of public water supplies began.
In the 1950's ADHD spread rapidly
among school children and gained much exposure in the medical
science and health literature. In 1963 the US PHS listed dozens of
symptoms associated with hyperactivity and officially changed the
name to "minimal brain dysfunction". By the the 1970's
some leading authorities noted that this disorder appeared to lie at
the root of nearly every type of childhood behaviour problem, and
had become the most commonly diagnosed illness among childhood
counsellors. (83,84)
In 1987 the American Medical
Association acknowledged that minimal brain damage had become the
leading disability reported by elementary schools, and "one of
the most common referral problems to psychiatry outpatients
clinics" (85)
Many studies on
thyroid hormones have shown that attention deficit and/or
hyperactivity disorders in children are linked to changes in the
levels of thyroid hormone in the blood, and that irritability
and aggressive behaviour are linked to thyroid hormone levels and
hypothyroidism. (86,87,88,89,90,91,92,93,94,95,96,97).
Behaviour disorders have been
associated with thyroid function for over 100 years.
In 1997 Aronson and Dodman
wrote, "the hypothyroid human patient has been reported to show
a wider range of behavioral symptoms. Particularly in the early
stages of the disease reduced cognitive function and concentration
together with impaired short-term memory may be confused with
attention deficit-hyperactivity disorder, and in one study 66%
of patients diagnosed with ADBD were found to be hypothyroid.
Supplementing their thyroid levels was largely curative. Visual and
auditory hallucinations may result from altered perception and have
been misdiagnosed as schizophrenia or psychosis. Other behavioral
symptoms have included fear - ranging from mild anxiety to frank
paranoia, mood swings and aggression."(98)
Many psychoactive drugs
including Prozac, Paxil and Luvox (Littleton) are fluorinated
medications. Rohypnol, the infamous date-rape drug, is fluorinated
Valium, which is about 20-30 times more potent than Valium alone. In
essence, these drugs effect enzyme functions in certain areas of the
brain to achieve the desired effect.(99)
Thyroid hormone disorders may
induce almost any psychiatric symptom or syndrome, including rage.
Peter Whybrow (100), of the University of
Pennsylvania, writes:
"An intimate association
between disturbances of thyroid hormone homeostasis and behavior has
been recognized for a long time already: Hyper- and hypothyroidism
can induce disturbances of mood and intellectual function (in severe
cases even psychosis can be mimicked). Reciprocally many psychiatric
disturbances, such as major depression and manic depressive disease
have associated with them disturbances of peripheral thyroid hormone
metabolism."
Whybrow reports on the
successful treatment of psychiatric disorder by supplementing T4 and
T3, both of which are reduced in plasma of rats after two months of
fluoride administration of 0.1 - 1mg/day.(101)
Recent Chinese studies show
that the influence of a high fluoride environment on intelligence
can occur early in development such as during the stages of
embryonic life or infancy when differentiation and growth are more
rapid. Ultramicroscopic study of embryonic brain tissue obtained
from termination of pregnancy operations in endemic fluorosis areas
showed "differentiation of brain nerve cells were poor, and
brain development was delayed."(102,103)
Highly alarming studies and
reviews in the last few years have documented the high accumulation
of fluorides in the fetus in countries all over the world.
(104,105,106,107)
Fluoride tends to fransfer
freely and immediately through the placenta, as has been shown in
numerous publications.(108,109)
It is important to note that
mother's milk passes on neglible amounts of fluoride in very high
fluoride-intake areas, as if "nature" meant to protect the
infant.(110)
THYROID/FLUORINE-IODINE ANTAGONISM
Additionally, a most important
factor to consider is the role of fluoride in iodine deficiency
disorders (IDD). The antagonistic relationship between fluoride and
iodine, being at opposite ends in the halogen group, has been
observed in many studies ever since Wagner von Jauregg began a mass
iodine-supplementation program in Austrian areas endemic with goiter
(enlargement of the thyroid gland) in the 1920's. (112) The late
George Waldbott (2) wrote that when the total iodine pool in the
body is low, fluoride interferes with the function of the thyroid
gland and thereby produces a fluoride-iodine antagonism, a view
shared and documented by numerous others. (113,114) However, it has
become clear within the last decade that fluoride excess, combined
with iodine _excess_ also exert "severe damage to the human
body". (115, 116) In the study by Yang et al.(116) on
children's intelligence in high iodine and fluorine regions, the
percentage of low-intelligence children was 16.7% at dental
fluorosis rates of 72.9%. This is comparable to fluorosis rates we
see in North America, some of which are up to 80%. (117)
A study published this year on
endemic goiter occurrences in the_absence_ of iodine deficiency
again showed higher goiter rates in high-fluoride areas in South
Africa.(118)
Could it be that the world-wide
"iodine deficiency" is actually fluoride excess? By
comparing IDD data supplied by the WHO (119) with fluorosis data
found on MEDLINE an answer may be found. You may judge for yourself:
|
COUNTRY
|
IDD/GOITER
|
FLUOROSIS
|
|
India
|
Very High (endemic)
|
Very High (Endemic)
|
|
Nigeria
|
High
|
High
|
|
Belgium
|
Moderately Low
|
Moderately Low
|
|
France
|
Low (3.9%)
|
Low (3%)
|
|
China
|
Very High (endemic)
|
Very High (endemic)
|
|
Mexico
|
Very High
(>60% San Luis Patosi)
|
Very High
(>60% San Luis Patosi)
|
|
Brazil
|
High (>30%)
|
High (>30%)
|
|
Italy
|
High (Mean 39%)
|
High (45% in fl.areas)
|
|
Tanzania
|
Very High (>60%)
|
Very High (60%)
|
|
Sudan
|
High
|
High
|
|
While it is well known that goiter and hypothyroidism
occur more often in mountainous areas, the same has now been shown
for dental fluorosis.(120,121)
[Note:While checking for IDD/Goiter data
for the US, we discovered that a national survey has never been
conducted. The only Canadian data available dates back 30 years, and
mentions earlier goiter occurences in the Great Lakes area.
(Brantford (Great Lakes) was the first Canadian city to be
fluoridated.))
Meanwhile, "iodine
deficiency" is now recognized as the most common cause of
preventable brain damage and mental disability in the world today.
It affects the brain development of the fetus. All thyroid
disorders, including hypothyroidism, can develop already in the
fetus.
Regarding the findings by Dr.
Phyllis Mullenix (65), and her observation that those exposed to
fluorides before birth were born hyperactive and remained so
throughout their lives, it fits very neatly with existing research
on hypothyroidism:
"Hypothyroidism
that is present from birth is referred to as congenital
hypothyroidism (CH). In North America, CH occurs in about 1 in 4000
live-born babies. The majority (over 90%) of affected babies in
North America have a permanent, life-long type of CH".(122)
Another thyroid/fluoride
connection can be seen in Jennifer Luke's data (123) which has shown
that fluoride accumulates in the pineal gland and inhibits its
production of melatonin. Luke showed in test animals that this
inhibition causes an earlier onset of sexual maturity, an effect
already reported in humans as well in 1956, as part of the
Kingston/Newburgh study. In fluoridated Newburgh, young girls
experienced earlier onset of menstruation than girls in
non-fluoridated Kingston (124).
The early onset of sexual
puberty is a well established symptom of thyroid hormone
dysfunction. Usually patients with low thyroid hormones also have
deficient secretion of growth hormone, and may have deficient
secretion of the gonadotropins, called LH and FSH, which stimulate
puberty and reproduction, and ACTH, which is necessary for cortisol
and hydrocortisone secretion by the adrenal gland. (125)
[In the above context it
should be noted, that aluminum fluoride also _mimicks_ the
inhibitory action of melatonin.(126)]
Another symptom of an
underactive thyroid condition (or iodine deficiency?) - severe
growth disturbances - was observed in 1935 by DeEds and Thomas in
children in areas where the water contained F- at 1-2 ppm. (127)
OSTEOPOROSIS, ARTHRITIS, and other
bone disorders
Left undetected and untreated,
thyroid disorder can elevate cholesterol levels, cause long-term
organ complications and may lead to irregular menstrual cycles,
infertility and worsening osteoporosis.(128,129,130)
Fluorides _accumulate_ in your
body. For this reason, as mentioned before, a MCL (Maximum
Contaminant Level) must be set for fluoride in the drinking water to
avoid Crippling Skeletal Fluorosis (CSF).
The US PHS wrote in 1991 that
"fluoride increases the stability of the crystal lattice in
bone, but makes bone more brittle... the total quantity of fluoride
ingested is the single most important factor in determining the
clinical course of skeletal fluorosis; the severity of symptoms
correlates directly with the level and duration of
exposure."(131)
On page 6 of the same report
it states:"Fluoride in the drinking water may increase the risk
of elderly men and women breaking bones"..pages 56-57:
"The weight of evidence from these experiments suggests that
fluoride added to water can increase the risk of hip fracture in
both elderly women and men...If this effect is confirmed, it would
mean that hip fracture in the elderly would replace dental fluorosis
as the most sensitive endpoint of fluoride exposure".
Since then several more
studies have been published, all showing greater incidence of hip
fractures among the elderly in fluoridated areas. (132,133,134) The
elderly are also the population suffering greatest from
hypothyroidism.
To understand the implications of fluoride
in bone disorders:
If you drink 1 cup (6oz) of green/black tea
a day, with F- content of 5mg, you can expect Chronic Skeletal
Fluorosis to appear as follows (135):
(100lbs. person)
Phase 1:.............................within 5 years
(sporadic pain; stiffness in joints; osteosclerosis of pelvis and
vertebral column)
Phase 2:.............................after 10 years
(chronic joint pain; arthritic symptoms; slight calcification of
ligaments; increased osteoclerosis/cancellous bones; with/without
osteoporosis of long bones)
Phase 3 (crippling fluorosis).......after 23 years
(limitation of joint movement; calcification of ligaments/neck, vert.
Column; crippling deformities/spine major joints; muscle
wasting;neurological defects/compression of spinal chord).
Comparing intake levels as high as they are (12) with statistical
data, it must become clear that this is already happening to a
significant portion of the population.
CONCLUSION:
As argued by Dean Burk and the attorneys who
established the connection between cancer deaths and fluoridation,
there is a premise in logic which states that the most obvious cause
of an event must be taken as face value while one searches for
alternative possibilities. Because it can be documented that
fluorides were given as medication for hyperthyroid patients it
should be considered the OBVIOUS cause for hypothyroidism and other
thyroid-hormone function-related disorders, including ADHD,
arthritis, osteoporosis, etc., especially at intake levels as high
as they are.
Fluoride poisoning can be observed in large groups of
the population, in the form of hypothyroidism. In 1995 one
publication (see 127) on hypothyroidism reported that 41 percent of
women had fatigue for no obvious reason in the past year. Of these
women, 57 percent said they experience fatigue three or more times a
week. More than half of women (51 percent) had experienced three or
more symptoms commonly associated with hypothyroidism over the past
year.
Other symptoms/associations of
hypothyroidism include loss of libido, carpal tunnel syndrome,
arthritis, lupus, fibromyalgia, memory loss, etc. [For a more
complete list, please see (74)]
Dental fluorosis is the first
visible indicator that severe thyroid hormone dysfunction has
occurred and is occurring. It is NOT a mere cosmetic effect as the
dental profession would like us to believe. The evidence is
staggering.
We must take immediate action to protect our children's
mental and physical health from the ever-increasing fluoride intake.
Water fluoridation must be halted, all foods must be labelled for F-
content, and emissions by industry must be strictly regulated.
Overall fluoride intake must be
radically reduced.
PLEASE
advise responsibly regarding green tea.
Sincerely,
Andreas Schuld
Parents of Fluoride Poisoned Children (PFPC)
Vancouver, B.C., Canada
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